Physiol. Genomics AJP: Lung Cellular and Molecular Physiology
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Corrigendum for Estany et al., Physiol. Genomics 31 (2) 236-243.
Physiol. Genomics 35: 197, 2008; doi:10.1152/physiolgenomics.zh7-3276-corr.2008
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Physiological Genomics 35:197 (2008)
1094-8341/08 $8.00 © 2008 American Physiological Society

Corrigendum

Corrigendum

Volume 31, October, 2007

Estany J, Tor M, Villalba D, Bosch L, Gallardo D, Jiménez N, Altet L, Noguera JL, Reixach J, Amills M, Sánchez A. Association of CA repeat polymorphism at intron 1 of insulin-like growth factor (IGF-I) gene with circulating IGF-I concentration, growth, and fatness in swine. Physiol Genomics 31: 236–243, 2007. First published June 19, 2007; doi:10.1152/physiolgenomics. 00283.2006; http://physiolgenomics.physiology.org/cgi/content/full/31/2/236.

We would like to make an amendment to our article in which we describe a relationship between the length of one microsatellite located at the pig insulin-like growth factor 1 gene and plasma levels of this hormone. According to Winterø et al. [Assignment of the gene for porcine insulin-like growth factor 1 (IGF1) to chromosome 5 by linkage mapping. Anim Genet 25: 37–39, 1994], this microsatellite mapped to intron 1 of the pig IGF1 gene. In consequence, in our article we have described the location of this microsatellite as intronic. However, we have recently blasted the microsatellite primer sequences against the GenBank database, and, according to the anotation of sequences X64400 and X17638, this microsatellite in reality maps to the promoter of the pig IGF1 gene. This new mapping information gives more support to our hypothesis of a regulatory role for the IGF1 microsatellite on gene expression.





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